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Human & Experimental Toxicology
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Oxidant signalling in carcinogenesis: a commentary

S S Deshpande

Division of Cardiology, Department of Medicine, The Johns Hopkins University School of Medicine, 720 Rutland Avenue, Ross 1023, Baltimore, Maryland 21205, USA

K Irani

Division of Cardiology, Department of Medicine, The Johns Hopkins University School of Medicine, 720 Rutland Avenue, Ross 1023, Baltimore, Maryland 21205, USA; kirani{at}jhmi.edu

It is becoming increasingly evident that reactive oxygen species (ROS) act at different stages of carcinogenesis, and thus play multiple roles in oncogenesis. In addition to being mutagenic and initiating tumors, ROS or carcinogens that result in ROS generation may affect tumor promotion and progression through varied effects on growth promoting, growth inhibitory, or apoptotic signaling pathways.

Key Words: carcinogenesis • intracellular signaling • reactive oxygen species

Human & Experimental Toxicology, Vol. 21, No. 2, 63-64 (2002)
DOI: 10.1191/0960327102ht211oa
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